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The EEG reveals bilateral, synchronous, symmetrical 3 to 4 Hz spike-wave discharges on a normal background. Four main theories on the pathophysiology of TA seizures have been proposed: centrencephalic theory, thalamic clock theory, cortical theory, and corticoreticular theory. More recently, it has been suggested that the cortical focus theory of TA seziures bridges cortical and thalamic theories. The mechanisms underlying the generalized spike-wave discharges that characterize this seizure appears to involve thalamocortical circuitry which has been studied extensively in the generation of sleep spindles. Their clinical and EEG manifestations are syndrome-related. For treatment, valproic acid, ethosuximide and lamotrigine are first-line medications.

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